Which copper peptide was actually studied?
Every row cites its source. Where a cell reflects our own reading of the evidence rather than an external source, the row says so.
Copper peptide marketing pools evidence from distinct molecules. This table returns each headline finding to the compound that earned it.
| Name | What it is | Evidence attributed to it | Is it GHK-Cu? | Source |
|---|---|---|---|---|
| GHK-Cu (copper tripeptide-1) | Gly-His-Lys copper complex; the subject of this site | Everything in our study table marked GHK-Cu, including the 1994 ulcer RCT and the null laser RCT | Yes | source |
| AHK-Cu | Alanyl-histidyl-lysine copper, a different tripeptide complex | The 2007 ex vivo human hair-follicle elongation study every hair page quotes | No | source |
| PC1031 | A ProCyte copper binding peptide from the 1990s program | The 1993 fuzzy rat follicular-enlargement result compared to minoxidil | No (related program compound) | source |
| GHK (copper-free) | The bare tripeptide without copper | The emphysema gene-signature work, the bleomycin mouse study of 2017, the sleep-deprivation mouse study, and the GHK arm of ALAVAX | No (the peptide without its copper) | source |